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NotaPublicado: 31 Jul 2012 18:30 

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----- Original Message -----
From: <A
title=Nauitec@aol.com href=<<mailto:Nauitec@aol.com<<>Nauitec@aol.com
To: <A title=deco@decompression.org
Sent: Thursday, October 03, 2002 11:23 PM
Subject: Deco Workshop Abstracts
All, The following
abstracts were submitted by Richard Pyle and Mike Powell for the Modern
Decompression Strategies Workshop in Tampa this coming February. As the rest of
the abstracts arrive, I will post them on the list. Hope to see you
there. Best regards, Timothy R. Oleary NAUI Director Technical
Operations POB 3867 South Padre Island, Texas
78597 956-761-7986 956-761-6039
fax www.nauitec.com nauitec@aol.com THE GENERATION OF TISSUE
NUCLEI Michael R. Powell, PhD NASA, Johnson Space Center Houston, Texas
77058 The Haldane model of decompression sought to outright avoid a
decompression gas phase by never allowing supersaturations to rise above the
metastable limit for phase separation. In the early 1970s, Doppler bubble
detectors demonstrated that gas phase formation was not uncommon in
decompressions, even those free from decompression sickness. Rather than a few
micronuclei generated with muscle activity, one model of decompression bubble
formation invokes a constant supply of micronuclei formed from hydrodynamic
cavitation of extracellular fluid in tissues laid out in a power law
distribution (many small, some medium, and a few large nuclei). More
strenuous movement will shift this distribution upwards in size. The
distribution will relax (half time in approximately one hour) to a<<baseline
distribution.<< It is not expected that the distribution will be the same
in all individuals and the Laplace limit (critical radius) will vary with
surface tension. These factors would be a part of the explanation as to
why there are differences between individuals with respect to pressure
NERD'S PERSPECTIVE R.L. Pyle Department of Natural
Sciences Bishop Museum Honolulu, HI I first began intentionally
incorporating initial deep decompression stops in 1987, after it became evident
that such stops conducted to keep captured fish specimens alive led to reduced
levels of fatigue following decompression dives. Nearly a decade later, I
published a brief article describing my deep-stop method, which has since come
to be known among many decompression divers as<<Pyle Stops<<. Because the
purpose and practice of my method of deep decompression stops has been
occasionally misunderstood, I will describe the history and implementation of
this method in greater detail. I will also compare this deep-stop method
to various other methods that introduce initial decompression stops deeper than
what are required by typical compartment-based decompression models. In
recent years, another pattern has emerged involving dives to depths in excess of
110m. My diving companions and I have compiled a database of logged dives to
depths of 60-150 m. All of these dives were conducted using
electronically-controlled, mixed-gas rebreathers. Despite very similar patterns
of hydration, exertion, general decompression strategy (including deep stops),
and other factors, there is a somewhat stark distinction between a 0% DCS
incidence rate on dives to less than 110m, compared with a 33% incidence rate on
dives to greater than 110 m. Acknowledging lack of ri orous controls, the
empirical pattern is that, all other factors being generally similar, there
appears be a sharp increase in DCS likelihood for dives to depths in excess of
110 m, perhaps representing some sort of physiological

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